Einträge von Penton, David auf dem Publikationsserver

Penton, David, Vohra, Twinkle, Banki, Eszter, Wengi, Agnieszka, Weigert, Maria, Forst, Anna-Lena, Bandulik, Sascha, Warth, Richard und Loffing, Johannes (2020) Collecting system–specific deletion of Kcnj10 predisposes for thiazide- and low-potassium diet–induced hypokalemia. Kidney International 97 (6), S. 1208-1218. Volltext nicht vorhanden.

Penton, David, Czogalla, Jan, Wengi, Agnieszka, Himmerkus, Nina, Loffing-Cueni, Dominique, Carrel, Monique, Rajaram, Renuga Devi, Staub, Olivier, Bleich, Markus , Schweda, Frank und Loffing, Johannes (2016) Extracellular K+ rapidly controls NaCl cotransporter phosphorylation in the native distal convoluted tubule by Cl− -dependent and independent mechanisms. The Journal of Physiology 594 (21), S. 6319-6331. Volltext nicht vorhanden.

Martins, Joana Raquel , Penton, David, Peyronnet, Rémi, Arhatte, Malika, Moro, Céline, Picard, Nicolas , Kurt, Birgül, Patel, Amanda, Honoré, Eric und Demolombe, Sophie (2016) Piezo1-dependent regulation of urinary osmolarity. Pflügers Archiv - European Journal of Physiology 468 (7), S. 1197-1206. Volltext nicht vorhanden.

Monticone, Silvia, Hattangady, Namita G., Penton, David, Isales, Carlos M., Edwards, Michael A., Williams, Tracy A., Sterner, Christina, Warth, Richard , Mulatero, Paolo und Rainey, William E. (2013) A Novel Y152C KCNJ5 Mutation Responsible for Familial Hyperaldosteronism Type III. The Journal of Clinical Endocrinology & Metabolism 98 (11), E1861-E1865. Volltext nicht vorhanden.

Warth, Richard, Barhanin, Jacques, Hartmann, Michaela, Lesage, Florian, Lalli, Enzo, Sterner, Christina, Tegtmeier, Ines , Schweda, Frank , Penton, David, Tauber, Philipp und Bandulik, Sascha (2013) Severe Hyperaldosteronism in Neonatal Task3 Potassium Channel Knockout Mice Is Associated With Activation of the Intraadrenal Renin-Angiotensin System. Endocrinology 154 (8), S. 2712-2722. Volltext nicht vorhanden.

Beuschlein, Felix , Boulkroun, Sheerazed , Osswald, Andrea, Wieland, Thomas, Nielsen, Hang N, Lichtenauer, Urs D, Penton, David, Schack, Vivien R, Amar, Laurence , Fischer, Evelyn, Walther, Anett, Tauber, Philipp, Schwarzmayr, Thomas, Diener, Susanne, Graf, Elisabeth, Allolio, Bruno, Samson-Couterie, Benoit, Benecke, Arndt, Quinkler, Marcus, Fallo, Francesco, Plouin, Pierre-Francois, Mantero, Franco, Meitinger, Thomas , Mulatero, Paolo, Jeunemaitre, Xavier, Warth, Richard , Vilsen, Bente , Zennaro, Maria-Christina , Strom, Tim M und Reincke, Martin (2013) Somatic mutations in ATP1A1 and ATP2B3 lead to aldosterone-producing adenomas and secondary hypertension. Nature Genetics 45 (4), S. 440-444. Volltext nicht vorhanden.

Mulatero, Paolo, Tauber, Philipp, Zennaro, Maria-Christina , Monticone, Silvia, Lang, Katharina, Beuschlein, Felix , Fischer, Evelyn, Tizzani, Davide, Pallauf, Anna, Viola, Andrea, Amar, Laurence , Williams, Tracy Ann, Strom, Tim M., Graf, Elisabeth, Bandulik, Sascha, Penton, David, Plouin, Pierre-François, Warth, Richard , Allolio, Bruno, Jeunemaitre, Xavier, Veglio, Franco und Reincke, Martin (2012) KCNJ5 Mutations in European Families With Nonglucocorticoid Remediable Familial Hyperaldosteronism. Hypertension 59 (2), S. 235-240. Volltext nicht vorhanden.

Barhanin, Jacques, Warth, Richard, Zennaro, Maria-Christina, Lalli, Enzo, Lesage, Florian, Budde, Thomas, El Wakil, Abeer, Cong, Lu Dang , Reichold, Markus, Tauber, Philipp , Haubs, Sophia , Schweda, Frank , Bandulik, Sascha und Penton, David (2012) Task3 Potassium Channel Gene Invalidation Causes Low Renin and Salt-Sensitive Arterial Hypertension. Endocrinology 153 (10), S. 4740-4748. Volltext nicht vorhanden.

Reichold, Markus, Zdebik, Anselm A., Lieberer, Evelyn, Rapedius, Markus, Schmidt, Katharina, Bandulik, Sascha, Sterner, Christina, Tegtmeier, Ines, Penton, David, Baukrowitz, Thomas, Hulton, Sally-Anne, Witzgall, Ralph , Ben-Zeev, Bruria, Howie, Alexander J., Kleta, Robert, Bockenhauer, Detlef und Warth, Richard (2010) KCNJ10 gene mutations causing EAST syndrome (epilepsy, ataxia, sensorineural deafness, and tubulopathy) disrupt channel function. Proceedings of the National Academy of Sciences 107 (32), S. 14490-14495. Volltext nicht vorhanden.