Publikationen von Hamdani, Nazha

Pabel, Steffen, Knierim, Maria, Stehle, Thea, Alebrand, Felix, Paulus, Michael, Sieme, Marcel, Herwig, Melissa , Barsch, Friedrich , Körtl, Thomas, Pöppl, Arnold, Wenner, Brisca, Ljubojevic-Holzer, Senka, Molina, Cristina E., Dybkova, Nataliya, Camboni, Daniele, Fischer, Thomas H., Sedej, Simon , Scherr, Daniel, Schmid, Christof, Brochhausen, Christoph, Hasenfuß, Gerd, Maier, Lars S., Hamdani, Nazha, Streckfuss-Bömeke, Katrin und Sossalla, Samuel (2022) Effects of Atrial Fibrillation on the Human Ventricle. Circulation Research 130 (7), S. 994-1010.

Tangos, Melina, Budde, Heidi, Kolijn, Detmar, Sieme, Marcel, Zhazykbayeva, Saltanat, Lódi, Mária , Herwig, Melissa, Gömöri, Kamilla, Hassoun, Roua, Robinson, Emma Louise , Meister, Toni Luise , Jaquet, Kornelia, Kovács, Árpád, Mustroph, Julian, Evert, Katja, Babel, Nina , Fagyas, Miklós, Lindner, Diana , Püschel, Klaus, Westermann, Dirk, Mannherz, Hans Georg, Paneni, Francesco , Pfaender, Stephanie, Tóth, Attila, Mügge, Andreas, Sossalla, Samuel und Hamdani, Nazha (2022) SARS-CoV-2 infects human cardiomyocytes promoted by inflammation and oxidative stress. International Journal of Cardiology 362, S. 196-205. Volltext nicht vorhanden.

Pabel, Steffen, Hamdani, Nazha, Singh, Jagdeep und Sossalla, Samuel (2021) Potential Mechanisms of SGLT2 Inhibitors for the Treatment of Heart Failure With Preserved Ejection Fraction. Frontiers in Physiology 2021 (12), S. 1-11.

Pabel, Steffen, Hamdani, Nazha, Luedde, Mark und Sossalla, Samuel (2021) SGLT2 Inhibitors and Their Mode of Action in Heart Failure—Has the Mystery Been Unravelled? Current Heart Failure Reports 18, S. 315-328.

Hassoun, Roua, Budde, Heidi, Zhazykbayeva, Saltanat, Herwig, Melissa, Sieme, Marcel, Delalat, Simin, Mostafi, Nusratul, Gömöri, Kamilla, Tangos, Melina, Jarkas, Muhammad, Pabel, Steffen, Bruckmüller, Stefanie, Skrygan, Marina, Lódi, Mária, Jaquet, Kornelia, Sequeira, Vasco, Gambichler, Thilo, Remedios, Cris Dos, Kovács, Árpád, Mannherz, Hans Georg, Mügge, Andreas, Sossalla, Samuel und Hamdani, Nazha (2021) Stress activated signalling impaired protein quality control pathways in human hypertrophic cardiomyopathy. International Journal of Cardiology 344, S. 160-169. Volltext nicht vorhanden.

Budde, Heidi, Hassoun, Roua, Tangos, Melina, Zhazykbayeva, Saltanat, Herwig, Melissa, Varatnitskaya, Marharyta , Sieme, Marcel , Delalat, Simin, Sultana, Innas, Kolijn, Detmar, Gömöri, Kamilla, Jarkas, Muhammad, Lódi, Mária , Jaquet, Kornelia, Kovács, Árpád, Mannherz, Hans Georg, Sequeira, Vasco , Mügge, Andreas, Leichert, Lars I. , Sossalla, Samuel und Hamdani, Nazha (2021) The Interplay between S-Glutathionylation and Phosphorylation of Cardiac Troponin I and Myosin Binding Protein C in End-Stage Human Failing Hearts. Antioxidants 10 (7), S. 1134. Volltext nicht vorhanden.

Sossalla, Samuel , Pabel, Steffen und Hamdani, Nazha (2020) A mechanistic rationale for the investigation of SGLT2 inhibitors in HFpEF - Letter regarding the article 'Baseline Characteristics of Patients with Heart Failure with Preserved Ejection Fraction in the EMPEROR-Preserved Trial'. European Journal of Heart Failure 23, S. 841.

Sossalla, Samuel, Pabel, Steffen, Reetz, Florian, Dybkova, Nataliya, Shomroni, Orr, Salinas, Gabriela , Mustroph, Julian, Hammer, Karin P., Hasenfuss, Gerd, Hamdani, Nazha, Maier, Lars S. und Streckfuss-Bömeke, Katrin (2020) Long-term effects of empagliflozin on excitation-contraction-coupling in human induced pluripotent stem cell cardiomyocytes. Journal of Molecular Medicine 98, S. 1689-1700.

Reil, Jan‐Christian , Reil, Gert‐Hinrich , Kovács, Árpád, Sequeira, Vasco, Waddingham, Mark T., Lodi, Maria , Herwig, Melissa, Ghaderi, Shahrooz, Kreusser, Michael M., Papp, Zoltán, Voigt, Niels, Dobrev, Dobromir , Meyhöfer, Svenja, Langer, Harald F., Maier, Lars S., Linz, Dominik , Mügge, Andreas, Hohl, Mathias, Steendijk, Paul und Hamdani, Nazha (2020) CaMKII activity contributes to homeometric autoregulation of the heart: A novel mechanism for the Anrep effect. The Journal of Physiology 598 (15), S. 3129-3153. Volltext nicht vorhanden.

Kolijn, Detmar, Pabel, Steffen, Tian, Yanna, Lódi, Mária , Herwig, Melissa , Carrizzo, Albino , Zhazykbayeva, Saltanat, Kovács, Árpád, Fülöp, Gábor Á, Falcão-Pires, Inês, Reusch, Peter H, Linthout, Sophie Van, Papp, Zoltán, van Heerebeek, Loek, Vecchione, Carmine, Maier, Lars S , Ciccarelli, Michele , Tschöpe, Carsten, Mügge, Andreas, Bagi, Zsolt, Sossalla, Samuel und Hamdani, Nazha (2020) Empagliflozin improves endothelial and cardiomyocyte function in human heart failure with preserved ejection fraction via reduced pro-inflammatory-oxidative pathways and protein kinase Gα oxidation. Cardiovascular Research 117 (2), S. 495-507. Volltext nicht vorhanden.

Loescher, Christine M., Breitkreuz, Martin, Li, Yong, Nickel, Alexander, Unger, Andreas, Dietl, Alexander, Schmidt, Andreas, Mohamed, Belal A. , Kötter, Sebastian, Schmitt, Joachim P., Krüger, Marcus, Krüger, Martina, Toischer, Karl, Maack, Christoph, Leichert, Lars I., Hamdani, Nazha und Linke, Wolfgang A. (2020) Regulation of titin-based cardiac stiffness by unfolded domain oxidation (UnDOx). Proceedings of the National Academy of Sciences 117 (39), S. 24545-24556. Volltext nicht vorhanden.

Maack, Christoph, Eschenhagen, Thomas, Hamdani, Nazha, Heinzel, Frank R., Lyon, Alexander R., Manstein, Dietmar J., Metzger, Joseph, Papp, Zoltán, Tocchetti, Carlo G., Yilmaz, M. Birhan, Anker, Stefan D., Balligand, Jean-Luc, Bauersachs, Johann, Brutsaert, Dirk, Carrier, Lucie, Chlopicki, Stefan , Cleland, John G., de Boer, Rudolf A., Dietl, Alexander, Fischmeister, Rodolphe, Harjola, Veli-Pekka, Heymans, Stephane, Hilfiker-Kleiner, Denise, Holzmeister, Johannes, de Keulenaer, Gilles, Limongelli, Giuseppe, Linke, Wolfgang A., Lund, Lars H., Masip, Josep, Metra, Marco, Mueller, Christian, Pieske, Burkert, Ponikowski, Piotr, Ristić, Arsen, Ruschitzka, Frank, Seferović, Petar M., Skouri, Hadi, Zimmermann, Wolfram H. und Mebazaa, Alexandre (2018) Treatments targeting inotropy. European Heart Journal 40 (44), S. 3626-3644. Volltext nicht vorhanden.