Einträge von Iberl, Sabine auf dem Publikationsserver

Mayer, Moritz N.S., Köhler, Lisa M., Paulus, Michael G. , Iberl, Sabine, Heinrich, Maria, Wagner, Stefan , Maier, Lars S. und Dietl, Alexander (2025) MATLAB-based Methods Allow Precise, High-Throughput Quantification of Nuclear Morphology and Texture in Tachycardiomyopathy. Journal of Histochemistry & Cytochemistry 73 (3-4), S. 115-128.

Wolf, Marietta, Brochhausen, Christoph , Ramakrishnan, Vignesh , Iberl, Sabine, Roth, Jonas, Seitz, Stephan, Burkhardt, Ralph und Stadler, Sonja C. (2024) Histologic Characterization of Tumor-Adjacent Mammary Adipose Tissue in Normal-Weight and Overweight/Obese Patients with Triple-Negative Breast Cancer. Cancers 16 (20), S. 3515.

Hammon, Kathrin, Renner, Kathrin, Althammer, Michael, Voll, Florian, Babl, Nathalie, Decking, Sonja-Maria, Siska, Peter J. , Matos, Carina, Conejo, Zugey Elizabeth Cárdenas, Mendes, Karina , Einwag, Friederike, Siegmund, Heiko, Iberl, Sabine, Berger, Raffaela S. , Dettmer, Katja, Schoenmehl, Rebecca, Brochhausen, Christoph , Herr, Wolfgang, Oefner, Peter J. , Rehli, Michael , Thomas, Simone und Kreutz, Marina (2024) D-2-hydroxyglutarate supports a tolerogenic phenotype with lowered major histocompatibility class II expression in non-malignant dendritic cells and acute myeloid leukemia cells. Haematologica.

Schelker, Roland Christian, Kratzer, Andrea Christina, Müller, Gunnar, Brochhausen, Christoph , Hart, Christina, Stempfl, Thomas, Heudobler, Daniel, Moehle, Christoph, Herr, Wolfgang, Iberl, Sabine und Grassinger, Jochen (2021) Stanniocalcin 1 is overexpressed in multipotent mesenchymal stromal cells from acute myeloid leukemia patients. Hematology 26 (1), S. 565-576.

Johannesen, Siw Wollebæk, Huie, J. Russell, Budeus, Bettina, Peters, Sebastian, Wirth, Anna M., Iberl, Sabine, Kammermaier, Tina, Kobor, Ines, Wirkert, Eva, Küspert, Sabrina, Tahedl, Marlene, Grassinger, Jochen, Pukrop, Tobias, Schneider, Armin, Aigner, Ludwig, Schulte-Mattler, Wilhelm , Schuierer, Gerhard, Koch, Winfried, Bruun, Tim-Henrik, Ferguson, Adam R. und Bogdahn, Ulrich (2021) Modeling and Bioinformatics Identify Responders to G-CSF in Patients With Amyotrophic Lateral Sclerosis. Frontiers in Neurology 12 (616289), S. 1-5.

Blücher, Christina, Iberl, Sabine, Schwagarus, Nancy, Müller, Silvana, Liebisch, Gerhard , Höring, Marcus, Hidrobo, Maria Soledad, Ecker, Josef, Spindler, Nick, Dietrich, Arne, Burkhardt, Ralph und Stadler, Sonja C. (2020) Secreted Factors from Adipose Tissue Reprogram Tumor Lipid Metabolism and Induce Motility by Modulating PPARα/ANGPTL4 and FAK. Molecular Cancer Research 18 (12), S. 1849-1862. Volltext nicht vorhanden.

Iberl, Sabine, Meyer, Anne-Louise, Müller, Gunnar, Peters, Sebastian, Johannesen, Siw, Kobor, Ines, Beier, Fabian , Brümmendorf, Tim H., Hart, Christina, Schelker, Roland, Herr, Wolfgang, Bogdahn, Ulrich und Grassinger, Jochen (2019) Effects of continuous high-dose G-CSF administration on hematopoietic stem cell mobilization and telomere length in patients with amyotrophic lateral sclerosis – a pilot study. Cytokine 120, S. 192-201. Volltext nicht vorhanden.

Johannesen, Siw Wollebæk , Budeus, Bettina, Peters, Sebastian, Iberl, Sabine, Meyer, Anne-Louise, Kammermaier, Tina, Wirkert, Eva, Bruun, Tim-Henrik, Samara, Verena C., Schulte-Mattler, Wilhelm, Herr, Wolfgang, Schneider, Armin, Grassinger, Jochen und Bogdahn, Ulrich (2018) Biomarker Supervised G-CSF (Filgrastim) Response in ALS Patients. Frontiers in Neurology 9 (971), S. 1-15.

Schelker, Roland Christian, Iberl, Sabine, Müller, Gunnar, Hart, Christina, Herr, Wolfgang und Grassinger, Jochen (2018) TGF-β1 and CXCL12 modulate proliferation and chemotherapy sensitivity of acute myeloid leukemia cells co-cultured with multipotent mesenchymal stromal cells. Hematology 23 (6), S. 337-345. Volltext nicht vorhanden.

Peters, Sebastian, Zitzelsperger, Eva, Kuespert, Sabrina, Iberl, Sabine, Heydn, Rosmarie, Johannesen, Siw, Petri, Susanne, Aigner, Ludwig, Thal, Dietmar R. , Hermann, Andreas, Weishaupt, Jochen H., Bruun, Tim-Henrik und Bogdahn, Ulrich (2017) The TGF-β System As a Potential Pathogenic Player in Disease Modulation of Amyotrophic Lateral Sclerosis. Frontiers in Neurology 8. Volltext nicht vorhanden.