Anzahl der Einträge: 9.
Artikel
Cigliano, Antonio,
Zhang, Shanshan,
Ribback, Silvia,
Steinmann, Sara,
Sini, Marcella,
Ament, Cindy E.,
Utpatel, Kirsten,
Song, Xinhua,
Wang, Jingxiao ,
Pilo, Maria G.,
Berger, Fabian,
Wang, Haichuan ,
Tao, Junyan,
Li, Xiaolei,
Pes, Giovanni M. ,
Mancarella, Serena,
Giannelli, Gianluigi,
Dombrowski, Frank,
Evert, Matthias,
Calvisi, Diego F.,
Chen, Xin und
Evert, Katja
(2022)
The Hippo pathway effector TAZ induces intrahepatic cholangiocarcinoma in mice and is ubiquitously activated in the human disease.
Journal of Experimental & Clinical Cancer Research 41, art.no.192.
Che, Li,
Paliogiannis, Panagiotis,
Cigliano, Antonio ,
Pilo, Maria G.,
Chen, Xin und
Calvisi, Diego F.
(2019)
Pathogenetic, Prognostic, and Therapeutic Role of Fatty Acid Synthase in Human Hepatocellular Carcinoma.
Frontiers in Oncology 9 (1412), S. 1-10.
Che, Li,
Chi, Wenna,
Qiao, Yu,
Zhang, Jie,
Song, Xinhua,
Liu, Ye,
Li, Lei,
Jia, Jiaoyuan,
Pilo, Maria G.,
Wang, Jingxiao,
Cigliano, Antonio ,
Ma, Zhilong,
Kuang, Wenhua,
Tang, Zefang,
Zhang, Zemin,
Shui, Guanghou,
Ribback, Silvia,
Dombrowski, Frank,
Evert, Matthias,
Pascale, Rosa Maria,
Cossu, Carla,
Pes, Giovanni Mario ,
Osborne, Timothy F.,
Calvisi, Diego F.,
Chen, Xin und
Chen, Ligong
(2019)
Cholesterol biosynthesis supports the growth of hepatocarcinoma lesions depleted of fatty acid synthase in mice and humans.
Gut 69 (1), S. 177-186.
Volltext nicht vorhanden.
Song, Xinhua,
Liu, Xianqiong,
Wang, Haichuan ,
Wang, Jingxiao ,
Qiao, Yu,
Cigliano, Antonio,
Utpatel, Kirsten,
Ribback, Silvia,
Pilo, Maria G.,
Serra, Marina,
Gordan, John D.,
Che, Li,
Zhang, Shanshan,
Cossu, Antonio,
Porcu, Alberto,
Pascale, Rosa M.,
Dombrowski, Frank,
Hu, Hongbo,
Calvisi, Diego F.,
Evert, Matthias und
Chen, Xin
(2018)
Combined CDK4/6 and Pan-mTOR Inhibition Is Synergistic Against Intrahepatic Cholangiocarcinoma.
Clinical Cancer Research 25 (1), S. 403-413.
Volltext nicht vorhanden.
Cigliano, Antonio ,
Wang, Chunmei,
Pilo, Maria G.,
Szydlowska, Marta ,
Brozzetti, Stefania ,
Latte, Gavinella,
Pes, Giovanni M.,
Pascale, Rosa M.,
Seddaiu, Maria A.,
Vidili, Gianpaolo,
Ribback, Silvia,
Dombrowski, Frank,
Evert, Matthias,
Chen, Xin und
Calvisi, Diego F.
(2017)
Inhibition of HSF1 suppresses the growth of hepatocarcinoma cell lines <i>in vitro</i> and AKT-driven hepatocarcinogenesis in mice.
Oncotarget 8 (33).
Volltext nicht vorhanden.
Che, Li,
Pilo, Maria G.,
Cigliano, Antonio ,
Latte, Gavinella ,
Simile, Maria M.,
Ribback, Silvia,
Dombrowski, Frank,
Evert, Matthias,
Chen, Xin und
Calvisi, Diego F.
(2017)
Oncogene dependent requirement of fatty acid synthase in hepatocellular carcinoma.
Cell Cycle 16 (6), S. 499-507.
Volltext nicht vorhanden.
Zhang, Shanshan,
Song, Xinhua,
Cao, Dan,
Xu, Zhong,
Fan, Biao,
Che, Li,
Hu, Junjie,
Chen, Bin,
Dong, Mingjie,
Pilo, Maria G.,
Cigliano, Antonio ,
Evert, Katja,
Ribback, Silvia,
Dombrowski, Frank,
Pascale, Rosa M.,
Cossu, Antonio,
Vidili, Gianpaolo,
Porcu, Alberto,
Simile, Maria M.,
Pes, Giovanni M.,
Giannelli, Gianluigi,
Gordan, John,
Wei, Lixin,
Evert, Matthias,
Cong, Wenming,
Calvisi, Diego F. und
Chen, Xin
(2017)
Pan-mTOR inhibitor MLN0128 is effective against intrahepatic cholangiocarcinoma in mice.
Journal of Hepatology 67 (6), S. 1194-1203.
Volltext nicht vorhanden.
Wang, Chunmei,
Che, Li,
Hu, Junjie,
Zhang, Shanshan,
Jiang, Lijie,
Latte, Gavinella ,
Demartis, Maria I.,
Tao, Junyan,
Gui, Bing,
Pilo, Maria G.,
Ribback, Silvia,
Dombrowski, Frank,
Evert, Matthias,
Calvisi, Diego F. und
Chen, Xin
(2016)
Activated mutant forms of PIK3CA cooperate with RasV12 or c-Met to induce liver tumour formation in mice via AKT2/mTORC1 cascade.
Liver International 36 (8), S. 1176-1186.
Volltext nicht vorhanden.
Hu, Junjie,
Che, Li,
Li, Lei,
Pilo, Maria G.,
Cigliano, Antonio ,
Ribback, Silvia,
Li, Xiaolei,
Latte, Gavinella ,
Mela, Marta,
Evert, Matthias,
Dombrowski, Frank,
Zheng, Guohua,
Chen, Xin und
Calvisi, Diego F.
(2016)
Co-activation of AKT and c-Met triggers rapid hepatocellular carcinoma development via the mTORC1/FASN pathway in mice.
Scientific Reports 6 (1).
Volltext nicht vorhanden.
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